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It is a 3-in-1 reference ebook. It supplies a whole clinical dictionary overlaying 1000's of phrases and expressions when it comes to aldosterone. It additionally provides vast lists of bibliographic citations. ultimately, it presents info to clients on tips on how to replace their wisdom utilizing numerous web assets. The e-book is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with learn devoted to aldosterone. in the event that your time is efficacious, this publication is for you. First, you won't waste time looking the net whereas lacking loads of suitable info. moment, the ebook additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing hundreds of thousands of web content.
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It is a 3-in-1 reference ebook. It supplies an entire scientific dictionary masking 1000's of phrases and expressions in relation to Cushing's disorder. It additionally supplies vast lists of bibliographic citations. eventually, it offers info to clients on how one can replace their wisdom utilizing a variety of web assets.
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Extra resources for Aldosterone - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Generate_Screen • Project Title: ESTROGEN AND SODIUM MODULATE HYPERTENSION IN AGING RATS Principal Investigator & Institution: Hinojosa-Laborde, Carmen; Associate Professor; Physiology; University of Texas Hlth Sci Ctr San Ant 7703 Floyd Curl Dr San Antonio, Tx 78229 Timing: Fiscal Year 2002; Project Start 01-FEB-2002; Project End 31-JAN-2007 Summary: (provided by applicant): The Dahl salt sensitive (S) rat will be studied as an animal model of post-menopausal hypertension to investigate the mechanisms responsible for the hypertension.
Nashville, Tn 372036917 Timing: Fiscal Year 2002; Project Start 01-APR-1992; Project End 31-MAR-2007 Summary: Description (provided by applicant) Transforming growth factor-beta 1 (TGFbeta) has been implicated as a key molecule in fibrosis, and its expression is increased in numerous fibrotic conditions. The relationship of TGFbeta to other profibrotic mechanisms, such as angiotensin and plasminogen activator inhibitor-1 (PAI-1), and the differential role of infiltrating versus parenchymal cells are complex.
In Specific Aim #1, we will test the hypothesis that drug-resistant, low-renin hypertensive blacks normalize their blood pressure in response to treatment with small doses of agents that reduce ENaC function. We will also test for a synergistic effect of spironolactone with amiloride to lower blood pressure. In Specific Aim #2, we will test the hypothesis that the mechanism whereby the anti-ENaC drugs lower blood pressure is by reducing an intrinsically higher level of ENaC function, as evidenced by lower levels of renin and aldosterone at baseline and greater increments during treatment in responders.